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Targeted ultra-deep sequencing reveals recurrent and mutually exclusive mutations of cancer genes in blastic plasmacytoid dendritic cell neoplasm.

TitleTargeted ultra-deep sequencing reveals recurrent and mutually exclusive mutations of cancer genes in blastic plasmacytoid dendritic cell neoplasm.
Publication TypeJournal Article
Year of Publication2014
AuthorsStenzinger, A., Endris V., Pfarr N., Andrulis M., Jöhrens K., Klauschen F., Siebolts U., Wolf T., Koch P-S., Schulz M., Hartschuh W., Goerdt S., Lennerz J. K., Wickenhauser C., Klapper W., Anagnostopoulos I., & Weichert W.
JournalOncotarget
Volume5
Issue15
Pagination6404-13
Date Published2014 Aug 15
ISSN1949-2553
KeywordsAged, Aged, 80 and over, Blast Crisis, Dendritic Cells, DNA Mutational Analysis, Female, Hematologic Neoplasms, Humans, Male, Middle Aged, Mutation, Plasmacytoma, Skin Neoplasms
Abstract

Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is a rare haematopoietic malignancy characterized by dismal prognosis and overall poor therapeutic response. Since the biology of BPDCN is barely understood, our study aims to shed light on the genetic make-up of these highly malignant tumors. Using targeted high-coverage massive parallel sequencing, we investigated 50 common cancer genes in 33 BPDCN samples. We detected point mutations in NRAS (27.3% of cases), ATM (21.2%), MET, KRAS, IDH2, KIT (9.1% each), APC and RB1 (6.1%), as well as in VHL, BRAF, MLH1, TP53 and RET1 (3% each). Moreover, NRAS-, KRAS- and ATM-mutations were found to be mutually exclusive and we observed recurrent mutations in NRAS, IDH2, APC and ATM. CDKN2A deletions were detected in 27.3% of the cases followed by deletions of RB1 (9.1%), PTEN and TP53 (3% each). The mutual exclusive distribution of some mutations may point to different subgroups of BPDCN whose biological significance remains to be explored.

DOI10.18632/oncotarget.2223
Alternate JournalOncotarget
PubMed ID25115387
PubMed Central IDPMC4171639

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