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Publications

Κατάλογος ενδεικτικών δημοσιεύσεων του τμήματος Ιατρικής ΑΠΘ σε διεθνή επιστημονικά περιοδικά κατά τα τελευταία 5 έτη.

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Eleftheriadis, T., Pissas G., Liakopoulos V., & Stefanidis I. (2013).  Inhibition of indoleamine 2,3-dioxygenase not only blocks autoreactive B cell activation, but it also reduces production of antibodies in general: comment on the article by Pigott and Mandik-Nayak.. Arthritis Rheum. 65(7), 1951-2.
Eleftheriadis, T., Pissas G., Yiannaki E., Markala D., Arampatzis S., Antoniadi G., et al. (2013).  Inhibition of indoleamine 2,3-dioxygenase in mixed lymphocyte reaction affects glucose influx and enzymes involved in aerobic glycolysis and glutaminolysis in alloreactive T-cells.. Hum Immunol. 74(12), 1501-9.
Sounidaki, M., Pissas G., Eleftheriadis T., Antoniadi G., Golfinopoulos S., Liakopoulos V., et al. (2019).  Indoleamine 2,3-dioxygenase suppresses humoral alloimmunity via pathways that different to those associated with its effects on T cells.. Biomed Rep. 1(1), 1-5.
Eleftheriadis, T., Liakopoulos V., Antoniadi G., Stefanidis I., & Galaktidou G. (2011).  Indoleamine 2,3-dioxygenase is increased in hemodialysis patients and affects immune response to hepatitis B vaccination.. Vaccine. 29(12), 2242-7.
Eleftheriadis, T., Pissas G., Antoniadi G., Spanoulis A., Liakopoulos V., & Stefanidis I. (2014).  Indoleamine 2,3-dioxygenase increases p53 levels in alloreactive human T cells, and both indoleamine 2,3-dioxygenase and p53 suppress glucose uptake, glycolysis and proliferation.. Int Immunol. 26(12), 673-84.
Eleftheriadis, T., Pissas G., Antoniadi G., Liakopoulos V., & Stefanidis I. (2015).  Indoleamine 2,3-dioxygenase depletes tryptophan, activates general control non-derepressible 2 kinase and down-regulates key enzymes involved in fatty acid synthesis in primary human CD4+ T cells.. Immunology. 146(2), 292-300.
Eleftheriadis, T., Pissas G., Golfinopoulos S., Liakopoulos V., & Stefanidis I. (2022).  Indoleamine 2,3-dioxygenase controls purinergic receptor-mediated ischemia-reperfusion injury in renal tubular epithelial cells.. J Basic Clin Physiol Pharmacol.
Eleftheriadis, T., Pissas G., Sounidaki M., Tsogka K., Antoniadis N., Antoniadi G., et al. (2016).  Indoleamine 2,3-dioxygenase, by degrading L-tryptophan, enhances carnitine palmitoyltransferase I activity and fatty acid oxidation, and exerts fatty acid-dependent effects in human alloreactive CD4+ T-cells.. Int J Mol Med. 38(5), 1605-1613.
Eleftheriadis, T., Pissas G., Liakopoulos V., & Stefanidis I. (2018).  Indoleamine 2, 3-dioxygenase Up-regulates Hypoxia-inducible Factor-1α Expression by Degrading L-tryptophan but Not Its Activity in Human Alloreactive T-cells.. Iran J Allergy Asthma Immunol. 17(1), 56-67.
Eleftheriadis, T., Pissas G., Remoundou M., Antoniadi G., Liakopoulos V., & Stefanidis I. (2013).  Increased visfatin in hemodialysis patients is associated with decreased demands for recombinant human erythropoietin.. Ren Fail. 35(10), 1399-403.
Eleftheriadis, T., Kartsios C., Pissas G., Liakopoulos V., Antoniadi G., Galaktidou G., et al. (2013).  Increased plasma angiogenin level is associated and may contribute to decreased T-cell zeta-chain expression in hemodialysis patients.. Ther Apher Dial. 17(1), 48-54.
Eleftheriadis, T., Pissas G., Antoniadi G., Tsogka K., Makri P., Liakopoulos V., et al. (2016).  Increased Indoleamine 2,3-Dioxygenase in Monocytes of Patients on Hemodialysis.. Iran J Kidney Dis. 10(2), 91-3.
Eleftheriadis, T., Pissas G., Mavropoulos A., Nikolaou E., Filippidis G., Liakopoulos V., et al. (2020).  In Mixed Lymphocyte Reaction, the Hypoxia-Inducible Factor Prolyl-Hydroxylase Inhibitor Roxadustat Suppresses Cellular and Humoral Alloimmunity.. Arch Immunol Ther Exp (Warsz). 68(6), 31.
Eleftheriadis, T., Sounidaki M., Pissas G., Antoniadi G., Liakopoulos V., & Stefanidis I. (2016).  In human alloreactive CD4⁺ T-cells, dichloroacetate inhibits aerobic glycolysis, induces apoptosis and favors differentiation towards the regulatory T-cell subset instead of effector T-cell subsets.. Mol Med Rep. 13(4), 3370-6.
Eleftheriadis, T., Pissas G., Liakopoulos V., & Stefanidis I. (2018).  IDO decreases glycolysis and glutaminolysis by activating GCN2K, while it increases fatty acid oxidation by activating AhR, thus preserving CD4+ T‑cell survival and proliferation.. Int J Mol Med. 42(1), 557-568.

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